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Argument for a 5G – COVID-19 Epidemic Causation Mechanism by Martin Pall, PhD

Update from Martin Pall, PhD, March 22, 2020

 

Two documents reprinted on [1], each argue that there are reasons to think that 5G radiation is greatly stimulating the coronavirus (COVID-19) pandemic and therefore, an important public health measure would be to shut down the 5G antennae and particularly the small cell 5G antennae in close proximity to our homes, schools, businesses, house of worship and hospitals.

 

The first of these documents [1] published by Miller et al., concerns the impact of 5G radiation on the immune system of the body and also suggests that 5G radiation may also increase the replication of the virus.  In both of these ways, 5G radiation may be expected to make the COVID-19 pandemic much worse.

 

The second of these documents [1] is my own and is derived from a larger document on 5G radiation effects [2].  It starts with the history of 5G in Wuhan, China, the epicenter of the COVID-19 epidemic.  Wuhan is China’s first 5G “smart city” and is the location of China’s first 5G highway where 5G radiation is being used to test self-driving vehicles.  Approximately 10,000 5G antennae were installed and activated in Wuhan in 2019, with approximately 75 to 80% of these installed and activated in the last 2 ½ months of the year.  The epidemic was first detected near the beginning of that 2 ½ month period and became vastly more severe, with extremely large increases in numbers of cases and in deaths by the end of 2019.  That may, of course be coincidental.  South Korea, which became the site of the worst epidemic outside of China, has large numbers of 5G antennae all over the country.  The Milan area of Italy, the worst epicenter in Europe also is a 5G center.  And Seattle area, the worst area in the U.S. is also a major 5G area.  Reports predict that New York City will shortly become the largest epicenter in the US is another 5G site.  These non-Chinese epidemic areas are not discussed in my paper, but these findings are accurate.  Again, the locations of these epicenters in 5G areas may be coincidental.

 

Electromagnetic fields, including the highly pulsed and therefore highly dangerous 5G millimeter wave radiation, act via activation of voltage-gated calcium channels (VGCCs) with VGCC activation producing five different effects, each of which have roles in stimulating the replication and spread of coronaviruses:

 

1.    Excessive intracellular calcium
2.    Oxidative stress
3.    NF-kappaB elevation
4.    Inflammation
5.    Apoptosis (programmed cell death)

 

The predominant cause of death in the COVID-19 epidemic, is pneumonia and each of these five effects also have roles in pneumonia, such that each of them is predicted to greatly increase the percent of people dying death in this epidemic.  It seems highly plausible that 5G radiation is greatly increasing the spread of the epidemic and also the death rate in individuals that are infected.

 

You may wish to consider all of this in conjunction with the broader findings with regard to the dangers of 5G and other effects apparently produced by 5G exposures [2].

 

How then did we get to this state?  Many independent scientists, including myself, have argued that there should be no 5G rollout until there is extensive biological safety testing of genuine 5G radiation with all of its dangerous modulating pulses.  However the industry has refused to get independent 5G testing and the FCC and other regulatory agencies have refused to require such testing.  Furthermore, the EMF “safety guidelines” which are supposed to protect us from health impacts of EMF radiation have been shown, based on eight different types of highly repeated studies, to fail massively to predict biological effects. They therefore fail to predict safety [3].  It follows from this that all assurances of safety based on these “safety guidelines” are fraudulent.  Consequently, there is no evidence whatsoever of 5G safety and much evidence of lack of safety.

 

It is my opinion, therefore, that 5G radiation is greatly stimulating the coronavirus (COVID-19) pandemic and also the major cause of death, pneumonia and therefore, an important public health measure would be to shut down the 5G antennae, particularly the small cell 5G antennae in close proximity to our homes, schools, businesses, houses of worship and hospitals.  I will list some of my professional qualifications following the citations.

 

Martin L. Pall,  Professor Emeritus of Biochemistry and Basic Medical Science, Washington State University

 

 

The VGCC activation mechanism has been amazingly well accepted in the scientific literature.
           My first (2013) paper on it was placed on the Global Medical Discovery web site as one of the top medical papers of 2013.  That paper has been cited 255 times according to the Google Scholar database.  Most new scientific paradigms are only slowly accepted and this is much, much faster than usual.
           I have given 59 invited professional talks on this topic in 15 countries, including 4 prestigious keynote addresses.  I had been scheduled to give 1 more prestigious keynote address in April (has been postponed because of COVID-19).
           Two of my papers, my neuropsychiatric paper and my Wi-Fi paper, are each described by the publishing journal as being the most often downloaded paper in the history of each journal – stunning scientific  interest in both papers.
           My recent talks, one sponsored by the Dept. of Engineering and Applied Science at Queens University and the other at the World Congress on Physics in Berlin (where I was given a certificate of recognition) show together that both engineers and physicists are starting to realize the importance of this mechanism.

 

Role of 5G in the Coronavirus Epidemic in Wuhan China

– by Martin Pall, PhD, Professor Emeritus, Washington State University

 

Wuhan, the capital of Hubei province in China, was chosen to be China’s first 5G “smart city” and the location of China’s first smart 5G highway. Wuhan is also the center of the horrendous coronavirus epidemic. The possible linkage between these two events was first discussed in an Oct. 31, 2019 article entitled: “Wuhan was the province where 5G was rolled out, now the center of deadly virus” https://5g-emf.com/wuhan-was-the-province-where-5g-was-rolled-out-now-the-center-of-deadly-virus/

 

The question that is being raised here is not whether 5G is responsible for the virus, but rather whether 5G radiation, acting via VGCC activation may be exacerbating the viral replication or the spread or lethality of the disease.  Let’s backtrack and look at the recent history of 5G in Wuhan in order to get some perspective on those questions.  An Asia Times article, dated Feb. 12, 2019 (https://www.asiatimes.com/2019/02/article/china-to-launch-first-5g-smart-highway) stated that there were 31 different 5G base stations (that is antennae) in Wuhan at the end of 2018. There were plans developed later such that approximately 10,000 5G antennae would be in place at the end of 2019, with most of those being on 5G LED smart street lamps.  The first such smart street lamp was put in place on May 14, 2019 (www.china.org.cn/china/2019-05/14/content_74783676.htm), but large numbers only started being put in place in October, 2019, such that there was a furious pace of such placement in the last 2 ½ months of 2019.  These findings show that the rapid pace of the coronavirus epidemic developed at least roughly as the number of 5G antennae became extraordinarily high.  So we have this finding that China’s 1st 5G smart city and smart highway is the epicenter of this epidemic and this finding that the epidemic only became rapidly more severe as the numbers of 5G antennae skyrocketed.

 

Are these findings coincidental or does 5G have some causal role in exacerbating the coronavirus epidemic?  In order to answer that question, we need to determine whether the downstream effects of VGCC activation exacerbate the viral replication, the effects of viral infection, especially those that have roles in the spread of the virus and also the mechanism by which this coronavirus causes death.

 

Accordingly, the replication of the viral RNA is stimulated by oxidative stress:

 

J Mol Biol. 2008 Nov 28;383(5):1081-96.   Variable oligomerization modes in coronavirus non-structural protein 9.  Ponnusamy R, Moll R, Weimar T, Mesters JR, Hilgenfeld R.

 

Other aspects of viral replication including those involved in the spread of the virus are stimulated by increased intracellular calcium [Ca2+]i, oxidative stress, NF-kappaB elevation, inflammation and apoptosis, each of which are increased following EMF exposure.  The first citation below shows an important role of VGCC activation in stimulating coronavirus infection.

 

Virology. 2020 Jan 2;539:38-48. Porcine deltacoronavirus (PDCoV) modulates calcium influx to favor viral replication.  Bai D, et al.

 

J Virol. 2011 May;85(9):4234-45. Distinct severe acute respiratory syndrome coronavirus-induced acute lung injury pathways in two different nonhuman primate species. Smits SL, et al.

 

Cell Calcium. 2018 Nov;75:30-41.  NAADP-dependent Ca2+ signaling regulates Middle East respiratory syndrome-coronavirus pseudovirus translocation through the endolysosomal system.  Gunaratne GS, et al.

 

J Virol. 2011 May;85(9):4234-45. Distinct severe acute respiratory syndrome coronavirus-induced acute lung injury pathways in two different nonhuman primate species. Smits SL, et al.

 

Proteome Sci. 2011 Mar 8;9:11. Proteomic analysis of chicken embryonic trachea and kidney tissues after infection in ovo by avian infectious bronchitis coronavirus.  Cao Z, et al.

 

Res Vet Sci. 2015 Jun;100:12-7.  Serum biomarkers of oxidative stress in cats with feline infectious peritonitis.  Tecles F, et al.

 

J Infect Dis. 2008 Mar 15;197(6):812-6.  Glucose-6-phosphate dehydrogenase deficiency enhances human coronavirus infection.  Wu YH et al.

 

J Virol. 1998 Jun;72(6):4918-24.  Transmissible gastroenteritis coronavirus induces programmed cell death in infected cells through a caspase-dependent pathway.  Eleouet JF, et al.

 

The predominant cause of death from this coronavirus is pneumonia.  Pneumonia is greatly exacerbated by each of those five downstream effects of VGCC activation, excessive intracellular calcium, oxidative stress, NF-kappaB elevation, inflammation and apoptosis. The first of the citations listed below shows that calcium channel blockers, the same type of drugs that block EMF effects, are useful in the treatment of pneumonia.  This predicts that EMFs, acting via VGCC activation, will produce increasingly severe pneumonia and therefore 5G radiation as well as other types of EMFs may well increase pneumonia deaths.

 

Zheng et al.  2016  Preadmission Use of Calcium Channel Blockers and Outcomes After Hospitalization With Pneumonia: A Retrospective Propensity-Matched Cohort Study.  Am J Ther. 2017 Jan/Feb;24(1):e30-e38.

 

Fang et al.  2017  Pneumolysin-Dependent Calpain Activation and Interleukin-1? Secretion in Macrophages Infected with Streptococcus pneumoniae.  Infect Immun. 2017 Aug 18;85(9). pii: e00201-17.

 

Fettel et al.  2019  Sphingosine-1-phosphate (S1P) induces potent anti-inflammatory effects in vitro and in vivo by S1P receptor 4-mediated suppression of 5-lipoxygenase activity.  FASEB J. 2019 Feb;33(2):1711-1726.

 

Liu and Shi.  2019  Calcium-activated chloride channel regulator 1 (CLCA1): More than a regulator of chloride transport and mucus production.  World Allergy Organ J. 2019 Nov 29;12(11):100077.

 

Medicine (Baltimore). 2018 Nov;97(45):e13087.  N-acetylcysteine improves oxidative stress and inflammatory response in patients with community acquired pneumonia: A randomized controlled trial. Zhang Q, et al.

 

Sci Rep. 2018 Oct 18;8(1):15393.  Surfactant protein D attenuates acute lung and kidney injuries in pneumonia-induced sepsis through modulating apoptosis, inflammation and NF-?B signaling. Du J, et al.

 

Curr Neurovasc Res. 2020 Jan 28.  MicroRNA (miR)-429 promotes inflammatory injury by targeting kruppel-like factor 4 (KLF4) in neonatal pneumonia.  Zhang L, et al.

 

Life Sci. 2019 Jul 1;228:189-197.  Long noncoding RNA SNHG16 targets miR-146a-5p/CCL5 to regulate LPS-induced WI-38 cell apoptosis and inflammation in acute pneumonia. Zhou Z, et al.

 

These all argue that 5G radiation is likely to greatly exacerbate the spread of the coronavirus and to greatly increase the lethality of the infections produced by it.  The good news is that it is likely that those of us that live in areas with no 5G radiation and who avoid other EMFs wherever possible will probably escape much of the impacts of this prospective global pandemic.  It is highly probable that one of the best things Wuhan can do to control the epidemic in the city is to turn off the 4G/5G system.

 

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